S produced and released by oligodendrocytes, thereby protecting the brain against
S produced and released by oligodendrocytes, thereby protecting the brain against extravascular hemoglobin toxicity [112]. Expression of haptoglobin is increased around the hematoma in animal models of ICH. Haptoglobin-deficient mice are more vulnerable to ICH-induced brain injury, and mice with haptoglobin overexpression are less susceptible to injury [112]. Sulforaphane, an NF-E2-related factor2 n-Phenylpiperazine-1-carboxamide (Nrf2) activator, …
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